Abstract
The effects of SR 31742A, a specific sigma site ligand, were investigated on regional neurotensin concentrations in rat brain. Both acute and chronic (21-day) treatment with either SR 31742A (20 mg/kg i.p.) or haloperidol (1 mg/kg i.p.) increased the neurotensin-like immunoreactivity in the nucleus accumbens. In contrast to haloperidol, the administration of SR 31742A failed to increase the concentration of neurotensin-like immunoreactivity in the caudate-putamen. Thus, the effects of SR 31742A appear to be selective for the limbic system.
MeSH terms
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Animals
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Antipsychotic Agents / pharmacology*
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Azepines / pharmacology*
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Caudate Nucleus / drug effects*
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Caudate Nucleus / metabolism
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Dose-Response Relationship, Drug
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Haloperidol / pharmacology
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Injections, Intraperitoneal
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Neurotensin / metabolism*
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Nucleus Accumbens / drug effects*
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Nucleus Accumbens / metabolism
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Putamen / drug effects*
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Putamen / metabolism
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Rats
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Rats, Sprague-Dawley
Substances
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Antipsychotic Agents
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Azepines
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3-(hexahydroazepin-1-yl)-1-(3-chloro-4-cyclohexylphenyl)-1-propene hydrochloride
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Neurotensin
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Haloperidol