Histamine-induced, central nervous system-mediated hyperglycemia is suppressed by atropine in the brain

Life Sci. 1993;52(15):PL107-10. doi: 10.1016/0024-3205(93)90096-l.

Abstract

We investigated the relationship between histamine and muscarinic cholinergic neurons in central nervous system (CNS)-mediated glucose regulation in anesthetized fed rats. The injection of pyrilamine (5 x 10(-7) mol) into the third cerebral ventricle suppressed the hyperglycemia induced by intraventricular injection of histamine (5 x 10(-7) mol). Ranitidine (5 x 10(-7) mol), however, did not suppress this hyperglycemia. The injection of atropine (5 x 10(-9)-5 x 10(-7) mol) into the third cerebral ventricle suppressed the histamine-induced hyperglycemia in a dose-dependent manner. These findings suggest that histamine induction of CNS-mediated hyperglycemia involves neuronal transmission not only via H1 receptors but also, at least in part, by muscarinic cholinergic neurons.

MeSH terms

  • Animals
  • Atropine / pharmacology*
  • Brain / drug effects*
  • Central Nervous System / physiopathology
  • Dose-Response Relationship, Drug
  • Histamine / administration & dosage
  • Histamine / pharmacology*
  • Histamine Antagonists / pharmacology*
  • Hyperglycemia / chemically induced*
  • Hyperglycemia / physiopathology
  • Injections, Intraventricular
  • Male
  • Neurotransmitter Agents / physiology
  • Pyrilamine / administration & dosage
  • Pyrilamine / pharmacology
  • Ranitidine / administration & dosage
  • Ranitidine / pharmacology
  • Rats
  • Rats, Wistar
  • Receptors, Muscarinic / physiology

Substances

  • Histamine Antagonists
  • Neurotransmitter Agents
  • Receptors, Muscarinic
  • Atropine
  • Histamine
  • Ranitidine
  • Pyrilamine