Monensin and forskolin inhibit the transcription rate of sucrase-isomaltase but not the stability of its mRNA in Caco-2 cells

FEBS Lett. 1993 Aug 9;328(1-2):55-8. doi: 10.1016/0014-5793(93)80964-v.

Abstract

Treatment of Caco-2 cells with forskolin (25 microM) or monensin (1 microM) has previously been shown to cause a marked decrease in the level of sucrase-isomaltase (SI) mRNA, without any effect on the expression of dipeptidylpeptidase IV (DPP-IV). In the present work, we report that there is no significant difference in the stability of SI mRNA between control and treated cells. On the other hand, we demonstrate a decrease in the transcription rate of SI mRNA which is sufficient to account for the decrease in the steady-state level of SI mRNA both in forskolin- and monensin-treated Caco-2 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Northern
  • Colforsin / pharmacology*
  • DNA Probes
  • Dipeptidyl Peptidase 4
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases / metabolism
  • Electrophoresis, Agar Gel
  • Gene Expression Regulation, Enzymologic / drug effects*
  • Humans
  • Monensin / pharmacology*
  • Oligo-1,6-Glucosidase / genetics
  • Oligo-1,6-Glucosidase / metabolism*
  • RNA, Messenger / metabolism*
  • Sucrase / genetics
  • Sucrase / metabolism*
  • Transcription, Genetic / drug effects*
  • Tumor Cells, Cultured

Substances

  • DNA Probes
  • RNA, Messenger
  • Colforsin
  • Monensin
  • Oligo-1,6-Glucosidase
  • Sucrase
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases
  • Dipeptidyl Peptidase 4