Induction and regulation of IL-8 and MCAF production in human brain tumor cell lines and brain tumor tissues

Eur Cytokine Netw. 1993 Sep-Oct;4(5):351-8.

Abstract

In order to elucidate the role of inflammatory cytokines in the central nervous system, we examined the production of two leukocyte chemoattractants, IL-8 and monocyte chemotactic and activating factor (MCAF) in brain tumor cell lines. The glioma cell lines tested exhibited high levels of IL-8 and MCAF mRNA expression upon stimulation with IL-1 or TNF-alpha, while none of the neuroblastoma cell lines expressed these cytokine mRNA. Both IL-8 and MCAF mRNA expression depended on the dose of IL-1 alpha and TNF-alpha and appeared very rapidly, reaching maximal levels at 3-6 hr, with substantial production of these cytokines in the culture supernatants. When various immunosuppressive drugs were tested, glucocorticoids but not other immunosuppressive drugs markedly inhibited the IL-1 or TNF-alpha-induced IL-8 and MCAF mRNA accumulation, suggesting that glucocorticoid is a potent regulator of these inflammatory cytokine production in the neural tissues. In addition, reverse transcription-polymerase chain reaction (RT-PCR) revealed the expression of IL-8 and MCAF mRNA expression in resected brain tumor tissues including glioblastoma, astrocytoma grade 2, ependymoma and medulloblastoma, indicating that these inflammatory cytokines are expressed in vivo.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Astrocytoma / immunology
  • Base Sequence
  • Brain Neoplasms / immunology*
  • Chemokine CCL2
  • Chemotactic Factors / biosynthesis*
  • Cytokines / biosynthesis*
  • Gene Expression Regulation, Neoplastic / drug effects
  • Glioblastoma / immunology
  • Glucocorticoids / pharmacology
  • Humans
  • Interleukin-8 / biosynthesis*
  • Kinetics
  • Molecular Sequence Data
  • Neuroblastoma / immunology
  • RNA, Messenger / biosynthesis
  • Tumor Cells, Cultured

Substances

  • Chemokine CCL2
  • Chemotactic Factors
  • Cytokines
  • Glucocorticoids
  • Interleukin-8
  • RNA, Messenger