Binding of [3H] SR 49059, a potent nonpeptide vasopressin V1a antagonist, to rat and human liver membranes

Biochem Biophys Res Commun. 1994 Feb 28;199(1):353-60. doi: 10.1006/bbrc.1994.1236.

Abstract

The new potent and selective nonpeptide vasopressin V1a antagonist, SR 49059, was tritiated and used for the characterization of rat and human liver AVP V1a receptors. Binding of [3H] SR 49059 was time-dependent, reversible and saturable. A single class of high affinity binding sites was identified with Kd values of 0.63 +/- 0.13 and 2.95 +/- 0.64 nM, in rat and human liver membranes, respectively. The maximal binding capacity (Bmax) was about 7 times higher in rat than in human liver preparations. The relative potencies of several AVP/oxytocin agonists or antagonists to inhibit [3H] SR 49059 binding confirmed that this ligand labeled a homogeneous population of sites with the expected AVP V1a profile. Furthermore, [3H] SR 49059 or unlabeled SR 49059 displayed only slight species differences between rat and human V1a receptors, whereas OPC-21268, another nonpeptide V1a antagonist, exhibited a high species-related potency with more than 500 fold higher affinity for rat than for human liver V1a receptors. Thus, [3H] SR 49059 is the first nonpeptide AVP V1a ligand reported having highly specific activity, stability, specificity and affinity. This makes it a suitable probe for labeling AVP V1a receptors in rat and also in human tissues.

MeSH terms

  • Animals
  • Arginine Vasopressin / antagonists & inhibitors*
  • Binding, Competitive
  • Cell Membrane / metabolism
  • Humans
  • Indoles / metabolism*
  • Indoles / pharmacology
  • Kinetics
  • Liver / metabolism*
  • Male
  • Pyrrolidines / metabolism*
  • Pyrrolidines / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Vasopressin / metabolism*

Substances

  • Indoles
  • Pyrrolidines
  • Receptors, Vasopressin
  • Arginine Vasopressin
  • relcovaptan