Auto-immune disease

Rheumatol Rehabil. 1976 Feb;15(1):1-9. doi: 10.1093/rheumatology/15.1.1.

Abstract

Auto-immune disease may result from the interaction of the genetic load of the individual, modification of self-tissue antigens by environmental agents such as virus or drugs and abnormalities of the immunological system itself such as the loss of controlling or suppressor T cells with age. In the majority of people the outcome is tolerance, maintenance of normal tissue architecture and function. In the unfortunate few the outcome is auto-immune disease, that is, failure to recognize "self".

Publication types

  • Review

MeSH terms

  • Animals
  • Antigen-Antibody Reactions
  • Autoimmune Diseases* / immunology
  • B-Lymphocytes / immunology
  • Freund's Adjuvant
  • Guinea Pigs
  • Haplorhini
  • Haptens
  • Humans
  • Immune Tolerance
  • Immunity, Cellular
  • Immunologic Deficiency Syndromes / immunology
  • Mice
  • Rats
  • T-Lymphocytes / immunology
  • Viruses / immunology

Substances

  • Haptens
  • Freund's Adjuvant