Absence of cytochrome c oxidase activity in a boy with dysfunction of renal tubules, brain and muscle

Eur J Pediatr. 1994 Apr;153(4):267-70. doi: 10.1007/BF01954517.

Abstract

We report on a boy who developed proximal renal tubular acidosis with loss of carnitine at the age of about 6 months. A few months later he began to suffer from progressive muscular weakness and neurological disturbances. Blood biochemistry showed elevated lactate and beta-hydroxybutyrate with increased lactate/pyruvate and beta-hydroxybutyrate/acetoacetate ratios. A high urinary excretion of lactate and citric acid cycle intermediates was found. These results indicated a defect of the mitochondrial respiratory chain. Analysis of biopsy material from skeletal muscle revealed low activities of all respiratory chain complexes. In muscle and fibroblasts cytochrome c-oxidase (complex IV) was absent. Despite high dose multi-vitamin therapy the boy died at the age of 30 months from central respiratory failure. At autopsy the neuropathological diagnosis of Leigh disease was made.

Publication types

  • Case Reports

MeSH terms

  • Acidosis, Renal Tubular / etiology*
  • Acidosis, Renal Tubular / metabolism
  • Brain Diseases / etiology
  • Brain Diseases / metabolism
  • Cytochrome-c Oxidase Deficiency*
  • Electron Transport Complex IV / metabolism
  • Fatal Outcome
  • Humans
  • Infant
  • Leigh Disease / blood
  • Leigh Disease / complications*
  • Leigh Disease / metabolism
  • Male
  • Mitochondrial Myopathies / etiology
  • Mitochondrial Myopathies / metabolism
  • Oxidoreductases / deficiency

Substances

  • Oxidoreductases
  • Electron Transport Complex IV