Conformation changes of p53 proteins in regulation of murine T lymphocyte proliferation

Cell Mol Biol Res. 1993;39(1):27-31.

Abstract

This report shows that p53 gene expression is required for proliferation of Concanavalin A (ConA)-stimulated murine T lymphocytes. p53 gene-specific antisense oligodeoxynucleotides strongly inhibited expression of p53 gene in T lymphocytes, causing suppression of ConA-induced cell proliferation. In contrast, the complementary-sense oligomers had no inhibitory effects. Northern and immunoprecipitation-blotting assay data, however, showed no change in p53 mRNA and proteins synthesized in T lymphocytes before and after ConA stimulation. p53 proteins in the resting and ConA-stimulated T cells had different immunoreactivity with specific monoclonal antibodies. p53 from resting T cells only reacted with "wild-type form" p53-specific monoclonal antibody (PAb246); whereas, p53 from ConA-stimulated T cells was recognized by "mutant-form" p53-specific monoclonal antibody (PAb421). These results suggest that the conformation of p53 proteins in resting and ConA-stimulated T cells are different. The p53 conformation changes may be related to the regulation of murine T lymphocyte proliferation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Division
  • Cells, Cultured
  • Concanavalin A / pharmacology
  • Gene Expression Regulation
  • Lymphocyte Activation / drug effects
  • Mice
  • Mice, Inbred BALB C
  • Oligonucleotides, Antisense / pharmacology
  • Protein Conformation*
  • T-Lymphocytes / immunology*
  • Tumor Suppressor Protein p53 / chemistry
  • Tumor Suppressor Protein p53 / physiology*

Substances

  • Oligonucleotides, Antisense
  • Tumor Suppressor Protein p53
  • Concanavalin A