Aminoglycoside antibiotics inhibit maxi-K+ channel in single isolated cochlear efferent nerve terminals

Hear Res. 1993 May;67(1-2):13-9. doi: 10.1016/0378-5955(93)90227-r.

Abstract

Patch clamp recordings were obtained from isolated cochlear efferent nerve terminals. The effect of aminoglycoside antibiotics on single maxi-K+ channels was determined. At positive voltages (cytosol with respect to extracellular side), neomycin, streptomycin, and kanamycin significantly reduced the single channel current amplitude of the maxi-K+ channel from the cytosolic side. The IC50 for neomycin was 9.10(-4) M from the cytosolic side and >> 10(-3) M from the extracellular side. Streptomycin and kanamycin were less potent. No significant difference in inhibition of the single channel current amplitude by 2.5.10(-4) M cytosolic neomycin was observed between 7.10(-4) M and 10(-6) M free cytosolic Ca2+. Neomycin had no significant effect on the open probability of the maxi-K+ channel either from the cytosolic or from the extracellular side. These findings demonstrate that the maxi-K+ channel in cochlear efferent nerve terminals can be a site of action for aminoglycoside antibiotics.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Anti-Bacterial Agents / pharmacology*
  • Cochlear Nerve / drug effects*
  • Electrophysiology
  • Gerbillinae
  • In Vitro Techniques
  • Kanamycin / pharmacology
  • Neomycin / pharmacology
  • Neurons, Efferent / drug effects*
  • Potassium Channels / drug effects*
  • Streptomycin / pharmacology

Substances

  • Anti-Bacterial Agents
  • Potassium Channels
  • Kanamycin
  • Neomycin
  • Streptomycin