Up-regulation of ETB receptor subtype mRNA by angiotensin II in rat cardiomyocytes

Biochem Biophys Res Commun. 1993 Aug 16;194(3):1282-7. doi: 10.1006/bbrc.1993.1962.

Abstract

The effect of angiotensin (ANG) II on the expression of endothelin (ET) receptor subtype (ETA, ETB) mRNA in cultured neonatal rat cardiomyocytes was studied. ANG II (10(-7) M) increased steady-state mRNA levels of ETB receptor, but not ETA receptor, during 6-12 hr incubation, whose effect was dose-dependent (10(-9)-10(-7) M). ANG II receptor type-1 (AT1) antagonist (CV-11974) completely inhibited the ANG II-induced up-regulation of ETB receptor, whereas the inhibitory effect by ANG II receptor type-2 (AT2) antagonist (PD 123319) was incomplete. These results suggest that ANG II up-regulates cardiac ETB receptor, predominantly via AT1 receptor.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Angiotensin II / pharmacology*
  • Animals
  • Animals, Newborn
  • Cells, Cultured
  • Dose-Response Relationship, Drug
  • Genetic Variation
  • Myocardium / cytology
  • Myocardium / metabolism*
  • RNA, Messenger / biosynthesis*
  • Rats
  • Rats, Wistar
  • Receptors, Endothelin / biosynthesis*
  • Up-Regulation*

Substances

  • RNA, Messenger
  • Receptors, Endothelin
  • Angiotensin II