Inhibition of DNA repair and sensitization of cisplatin in human ovarian carcinoma cells by interleukin-1 alpha

Biochem Biophys Res Commun. 1993 Aug 31;195(1):294-300. doi: 10.1006/bbrc.1993.2044.

Abstract

Interleukin-1 alpha induced an increase in both the cellular accumulation of cis-diamminedichloroplatinum (II) (cisplatin) and DNA platination and significantly reduced the removal of platinum from DNA of human ovarian (NIH: OVCAR-3) carcinoma cells in culture. The combinations of IL-1 alpha and cisplatin were highly synergistic against these ovarian carcinoma cells and maximum levels of sensitization (15-20-fold) were observed during simultaneous exposure of cisplatin and IL-1 alpha. IL-1 alpha specific receptor antagonist decreased this synergy. These results strongly indicate that IL-1 alpha inhibits DNA repair, and this inhibition of DNA repair may explain, in part, a strong synergistic interaction between IL-1 alpha and cisplatin in NIH: OVCAR-3 cells.

MeSH terms

  • Cell Line
  • Cell Survival / drug effects
  • Cisplatin / metabolism*
  • Cisplatin / toxicity*
  • DNA Repair / drug effects*
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Female
  • Humans
  • Interleukin-1 / toxicity*
  • Kinetics
  • Ovarian Neoplasms
  • Receptors, Interleukin-1 / antagonists & inhibitors
  • Recombinant Proteins / toxicity
  • Tumor Cells, Cultured

Substances

  • Interleukin-1
  • Receptors, Interleukin-1
  • Recombinant Proteins
  • Cisplatin