Up-regulation of polysialic acid in peripheral myelinated axons of rat chronically exposed to 2,5-hexanedione

Pharmacol Toxicol. 1993 Apr-May;72(4-5):236-9. doi: 10.1111/j.1600-0773.1993.tb01642.x.

Abstract

Myelinated nerve fibres isolated from Wistar rats chronically exposed to 2,5-hexanedione (0.8 ml/kg/day, intraperitoneally) over a period of 20 days, were stained with lectin-horseradish peroxidase conjugates. The lectins with high affinity for terminal D-galactopyranosyl residues, Bandeiraea simplicifolia-B4 (BSA I-B4) and peanut agglutinin (PNA), showed glycoconjugates in the control nodes of Ranvier. In the treated animals, application of PNA-HRP caused weak reactivity to the node of Ranvier; digestion with sialidase prior to the application of PNA-HRP conjugate enhanced reactivity, thus revealing the presence of a sialoglycoprotein. The results indicate that glycoconjugates of the Ranvier node undergo a rearrangement during exposure to 2,5-hexanedione. In particular, neutral glycoproteins with terminal galactose are replaced by sialoglycoproteins. These findings are consistent with the proposed role of polysialic acid as a regulator of axonal behaviour during regeneration.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Axons / metabolism*
  • Hexanones / toxicity*
  • Histocytochemistry
  • Horseradish Peroxidase
  • Lectins / metabolism
  • Male
  • Myelin Sheath / metabolism*
  • Polysaccharides / metabolism
  • Ranvier's Nodes / metabolism
  • Rats
  • Rats, Wistar
  • Sciatic Nerve / drug effects
  • Sialic Acids / metabolism*
  • Up-Regulation

Substances

  • Hexanones
  • Lectins
  • Polysaccharides
  • Sialic Acids
  • polysialic acid
  • 2,5-hexanedione
  • Horseradish Peroxidase