Antagonism of the inhibitory action of neuropeptide Y (NPY) in guinea-pig trachea by the C-terminal fragment NPY (2-36)

Br J Pharmacol. 1993 Aug;109(4):902-4. doi: 10.1111/j.1476-5381.1993.tb13705.x.

Abstract

The C-terminal fragment of neuropeptide Y (NPY), NPY(2-36) was used as a means of discriminating between two differently located NPY receptor sites in guinea-pig trachea. Both NPY and NPY(2-36) reduced the maximal relaxation elicited by vasoactive intestinal peptide (VIP). In contrast, the C-terminal fragment did not mimic the inhibitory action of NPY on the noradrenaline-(NA) evoked response. However, pretreatment of the trachea with 30 nM NPY(2-36), 5 min before generating NA and VIP concentration-response curves in the presence of NPY, abolished the inhibitory effect of NPY on NA-elicited response but did not affect the modulatory action of NPY on VIP-induced relaxation. These results suggest that the two differently located NPY receptor sites in guinea-pig trachea are of two distinct subpopulations.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Guinea Pigs
  • In Vitro Techniques
  • Isometric Contraction / drug effects
  • Muscle Relaxation / drug effects
  • Muscle, Smooth / drug effects*
  • Neuropeptide Y / antagonists & inhibitors*
  • Neuropeptide Y / pharmacology*
  • Norepinephrine / pharmacology
  • Peptide Fragments / pharmacology*
  • Receptors, Neuropeptide Y / drug effects
  • Trachea / drug effects
  • Vasoactive Intestinal Peptide / pharmacology

Substances

  • Neuropeptide Y
  • Peptide Fragments
  • Receptors, Neuropeptide Y
  • neuropeptide Y (2-36)
  • Vasoactive Intestinal Peptide
  • Norepinephrine