Interleukin 10 reduces the release of tumor necrosis factor and prevents lethality in experimental endotoxemia

J Exp Med. 1993 Feb 1;177(2):547-50. doi: 10.1084/jem.177.2.547.

Abstract

Because of its ability to efficiently inhibit in vitro cytokine production by activated macrophages, we hypothesized that interleukin (IL) 10 might be of particular interest in preventing endotoxin-induced toxicity. We therefore examined the effects of IL-10 administration before lipopolysaccharide (LPS) challenge in mice. A marked reduction in the amounts of LPS-induced tumor necrosis factor (TNF) release in the circulation was observed after IL-10 pretreatment at doses at low as 10 U. IL-10 also efficiently prevented the hypothermia generated by the injection of 100 micrograms LPS. Finally, pretreatment with a single injection of 1,000 U IL-10 completely prevented the mortality consecutive to the challenge with 500 micrograms LPS, a dose that was lethal in 50% of the control mice. We conclude that IL-10 inhibits in vivo TNF secretion and protects against the lethality of endotoxin in a murine model of septic shock.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Endotoxins / toxicity*
  • Hypothermia / prevention & control
  • Interleukin-10 / pharmacology*
  • Lipopolysaccharides / toxicity*
  • Mice
  • Mice, Inbred BALB C
  • Molecular Sequence Data
  • Oligodeoxyribonucleotides / chemistry
  • Secretory Rate / drug effects
  • Shock, Septic / prevention & control*
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Endotoxins
  • Lipopolysaccharides
  • Oligodeoxyribonucleotides
  • Tumor Necrosis Factor-alpha
  • Interleukin-10