Selective reversal of vinblastine resistance in multidrug-resistant cell lines by tamoxifen, toremifene and their metabolites

Eur J Cancer. 1993;29A(8):1152-7. doi: 10.1016/s0959-8049(05)80306-5.

Abstract

In this study we describe the effects of tamoxifen, toremifene and their 4-hydroxy and N-desmethyl metabolites on the toxicity of a range of drugs to human breast and lung cancer and to Chinese hamster ovary cell lines, determined using a tetrazolium-based semi-automated colorimetric assay. Vinblastine resistance was completely abolished in an mdr1-transfected lung cancer cell line (S1/1.1), indicating that P-glycoprotein-mediated multidrug resistance can be fully reversed by anti-oestrogens. A substantial (14- to 39-fold) enhancement of vinblastine toxicity to highly multidrug-resistant (MCF-7Adr) cells expressing P-glycoprotein was also observed in the presence of tamoxifen, toremifene and their metabolites, while m-amsacrine, cisplatin and melphalan toxicity was unaffected.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amsacrine / therapeutic use
  • Animals
  • Breast Neoplasms / drug therapy
  • CHO Cells / drug effects
  • Cell Survival / drug effects
  • Cisplatin / therapeutic use
  • Cricetinae
  • Dose-Response Relationship, Drug
  • Drug Resistance
  • Humans
  • Lung Neoplasms / drug therapy
  • Melphalan / therapeutic use
  • Tamoxifen / pharmacology*
  • Toremifene / pharmacology*
  • Tumor Cells, Cultured / drug effects
  • Vinblastine / therapeutic use*

Substances

  • Amsacrine
  • Tamoxifen
  • Vinblastine
  • Toremifene
  • Cisplatin
  • Melphalan