Constitutive and cytokine-induced expression of the melanoma growth stimulatory activity/GRO alpha gene requires both NF-kappa B and novel constitutive factors

J Biol Chem. 1995 Dec 22;270(51):30619-26. doi: 10.1074/jbc.270.51.30619.

Abstract

Melanoma growth stimulatory activity (MGSA)/growth regulated (GRO) and interleukin-8 (IL-8) are highly related chemokines that have a causal role in melanoma progression. Expression of these chemokines is similar in that both require the NF-kappa B element and additional regions such as the CAAT/enhancer binding protein (C/EBP) element of the IL-8 promoter. The constitutive and cytokine IL-1-induced promoter activity of the chemokine MGSA/GRO alpha in normal retinal pigment epithelial and the Hs294T melanoma cells is partially regulated through the NF-kappa B element, which binds both NF-kappa B p50 and RelA (NF-kappa B p65) homodimers and heterodimers. Mutational analysis of the MGSA/GRO alpha promoter reveals that, in addition to the NF-kappa B element, the immediate upstream region (IUR) is necessary for basal expression in retinal pigment epithelial and Hs294T cells. Gel mobility shift and UV cross-linking analyses demonstrate that several constitutive DNA binding proteins interact with the IUR. Although this region has sequence similarity to the several transcription factor elements including C/EBP, the IUR includes sequences that have no similarity to previously identified enhancer regions. Furthermore, RelA transactivates through either the NF-kappa B element or the IUR, suggesting a putative interaction between NF-kappa B and this novel complex.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Base Sequence
  • CCAAT-Enhancer-Binding Proteins
  • Cell Line
  • Chemokine CXCL1
  • Chemokines, CXC*
  • Chemotactic Factors / biosynthesis*
  • Chemotactic Factors / genetics
  • Cross-Linking Reagents
  • Cytokines / biosynthesis*
  • Cytokines / genetics
  • DNA Mutational Analysis
  • DNA-Binding Proteins / metabolism
  • Gene Expression* / drug effects
  • Growth Inhibitors / biosynthesis*
  • Growth Substances / biosynthesis*
  • Growth Substances / genetics
  • Humans
  • Intercellular Signaling Peptides and Proteins*
  • Interleukin-8 / genetics
  • Melanoma
  • Molecular Sequence Data
  • NF-kappa B / metabolism*
  • Nuclear Proteins / metabolism
  • Pigment Epithelium of Eye / metabolism*
  • Promoter Regions, Genetic
  • Sequence Homology, Nucleic Acid
  • Transcription Factor RelA
  • Transcription Factors
  • Tumor Cells, Cultured
  • Ultraviolet Rays

Substances

  • CCAAT-Enhancer-Binding Proteins
  • CXCL1 protein, human
  • Chemokine CXCL1
  • Chemokines, CXC
  • Chemotactic Factors
  • Cross-Linking Reagents
  • Cytokines
  • DNA-Binding Proteins
  • Growth Inhibitors
  • Growth Substances
  • Intercellular Signaling Peptides and Proteins
  • Interleukin-8
  • NF-kappa B
  • Nuclear Proteins
  • Transcription Factor RelA
  • Transcription Factors