Activated B cells express functional Fas ligand

Eur J Immunol. 1996 Mar;26(3):721-4. doi: 10.1002/eji.1830260332.

Abstract

Fas ligand (FasL, Apo-1L) is a member of the tumor necrosis factor protein family and binding to its receptor (Fas, Apo-1, CD95) triggers cell death through apoptosis. Ligand expression is restricted to cells with known cytolytic activity and found on hematopoietic cells of the T cell and natural killer lineage. Here we provide evidence that B lymphocytes can express FasL. Flow cytometric analysis revealed that FasL is expressed on the surface of B cells upon stimulation with either lipopolysaccharide or phorbol 12-myristate 13-acetate/ionomycin. FasL expression on activated B cells was confirmed by western blot and reverse transcriptase polymerase chain reaction analysis. FasL on B cells is functional since lipopolysaccharide-activated B lymphocytes derived from wild type, but not from gld mutant mice, were able to kill Fas-sensitive target cells. Our data suggest that the Fas system may contribute to the control of B cell homeostasis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • B-Lymphocytes / drug effects
  • B-Lymphocytes / immunology*
  • B-Lymphocytes / metabolism*
  • Base Sequence
  • Cytotoxicity, Immunologic / drug effects
  • Fas Ligand Protein
  • Ligands
  • Lipopolysaccharides / pharmacology
  • Lymphocyte Activation*
  • Membrane Glycoproteins / biosynthesis*
  • Membrane Glycoproteins / physiology*
  • Mice
  • Mice, Inbred BALB C
  • Molecular Sequence Data
  • Tumor Necrosis Factor-alpha / physiology
  • fas Receptor / biosynthesis
  • fas Receptor / physiology*

Substances

  • Fas Ligand Protein
  • Fasl protein, mouse
  • Ligands
  • Lipopolysaccharides
  • Membrane Glycoproteins
  • Tumor Necrosis Factor-alpha
  • fas Receptor