Dominant negative stat3 mutant inhibits interleukin-6-induced Jak-STAT signal transduction

J Biol Chem. 1996 Mar 15;271(11):5961-4. doi: 10.1074/jbc.271.11.5961.

Abstract

Interleukin-6 (IL-6) induces tyrosine phosphorylation and activation of the latent transcription factor Stat3 in HepG2 cells. Mutation of Stat3 tyrosine 705 to phenylalanine (Y705F) inhibits IL-6-induced tyrosine phosphorylation of this Stat3 mutant in transfected HepG2 cells. In cotransfections of HepG2 cells, the Stat3 mutant Y705F causes a reduction of the tyrosine phosphorylation of wild type Stat3-FLAG. Moreover, Y705F inhibits the action of endogenous Stat3 in cotransfected cells, reducing IL-6 induction of a Stat3-responsive reporter construct. Y705F therefore acts as a dominant negative mutation of Stat3.

MeSH terms

  • Amino Acid Sequence
  • Cell Line
  • DNA-Binding Proteins / genetics*
  • DNA-Binding Proteins / metabolism
  • Humans
  • Interleukin-6 / pharmacology*
  • Models, Genetic
  • Molecular Sequence Data
  • Mutation*
  • Oligopeptides
  • Peptides / genetics
  • Phosphorylation
  • STAT3 Transcription Factor
  • Signal Transduction / genetics*
  • Trans-Activators / genetics*
  • Trans-Activators / metabolism
  • Transfection
  • Tyrosine / genetics
  • Tyrosine / metabolism

Substances

  • DNA-Binding Proteins
  • Interleukin-6
  • Oligopeptides
  • Peptides
  • STAT3 Transcription Factor
  • STAT3 protein, human
  • Trans-Activators
  • Tyrosine
  • FLAG peptide