Improvement of insulin sensitivity by metformin treatment does not lower blood pressure of nonobese insulin-resistant hypertensive patients with normal glucose tolerance

J Clin Endocrinol Metab. 1996 Apr;81(4):1568-74. doi: 10.1210/jcem.81.4.8636369.

Abstract

Nine hypertensive patients with body mass indexes between 24-27 kg/m2 and normal glucose tolerance with at least a postchallenge plasma insulin level greater than 360 pmol/L were recruited for a double blind, cross-over study with metformin (850 mg, twice daily) and placebo. Each treatment lasted 1 month. Before and after each treatment, hormone and substrate concentrations were determined, blood pressure was monitored over 24 h, and insulin sensitivity was measured by a euglycemic (4.7 mmol/L) hyperinsulinemic (450 pmol/L) clamp study. Renal cation excretion and erythrocyte membrane cation heteroexchange were measured. Metformin, compared to placebo, did not affect body weight (70 +/- 7 vs. 70 +/- 7 kg), fasting plasma glucose (4.8 +/- 0.1 vs. 4.8 +/- 0.1 mmol/L), total cholesterol (5.38+/0.33 vs. 5.48 +/- 0.38 mmol/L), or triglycerides (1.73 +/- 0.72 vs. 1.91 0.89 mmol/L). Nevertheless, after metformin treatment, the plasma high density lipoprotein cholesterol concentration increased (1.42 +/- 0.18 vs. 1.34 0.16 mmol/L), and the plasma insulin level dropped (62 +/- 10 vs. 88+/- 12 pmol/L; both P < 0.05). Insulin-mediated glucose disposal was higher after metformin treatment (26.1 +/- 2.4 vs. 19.3 +/- 2.3 micromol/min x kg; P < 0.01), whereas hepatic glucose production was completely suppressed. These positive metformin-induced metabolic effects were not associated with a significant change in mean daily blood pressure levels (141 +/- 6/89 +/- 3 vs. 142 +/- 7/90 +/- 3 mm Hg). Compared to placebo, metformin increased the excretion of sodium, potassium, and lithium by enhancing their glomerular filtration rate. Na+/Li+ countertransport was not affected by metformin. However, the apparent affinity for H+ of Na+/H+ exchange was increased, and the Hill coefficient was decreased. In conclusion, 1 month of metformin administration to patients with essential hypertension and normal glucose tolerance 1) reduces the basal plasma insulin concentration, 2) improves whole body insulin-mediated glucose utilization, and 3) improves plasma high density lipoprotein cholesterol levels. Despite these positive effects, metformin did not reduce arterial blood pressure.

Publication types

  • Clinical Trial
  • Comparative Study
  • Randomized Controlled Trial

MeSH terms

  • Adult
  • Aged
  • Alanine / blood
  • Aldosterone / blood
  • Aldosterone / urine
  • Blood Glucose / drug effects
  • Blood Glucose / metabolism
  • Blood Pressure / drug effects*
  • Body Weight
  • Cross-Over Studies
  • Double-Blind Method
  • Epinephrine / blood
  • Erythrocyte Membrane / drug effects
  • Erythrocyte Membrane / metabolism
  • Female
  • Glucose / metabolism
  • Glucose Clamp Technique
  • Glycerol / blood
  • Humans
  • Hypertension / blood
  • Hypertension / physiopathology*
  • Hypoglycemic Agents / therapeutic use*
  • Insulin / administration & dosage
  • Insulin / pharmacology*
  • Insulin Resistance*
  • Ketone Bodies / blood
  • Kidney / physiopathology
  • Lactates / blood
  • Male
  • Metformin / therapeutic use*
  • Middle Aged
  • Norepinephrine / blood
  • Placebos
  • Renin / blood
  • Sodium / metabolism

Substances

  • Blood Glucose
  • Hypoglycemic Agents
  • Insulin
  • Ketone Bodies
  • Lactates
  • Placebos
  • Aldosterone
  • Metformin
  • Sodium
  • Renin
  • Glucose
  • Alanine
  • Glycerol
  • Norepinephrine
  • Epinephrine