In vivo role of IL-10 and IL-12 during development of Sjögren's syndrome in MRL/lpr mice

Cell Immunol. 1996 Mar 15;168(2):243-50. doi: 10.1006/cimm.1996.0072.

Abstract

Expression of local cytokine genes including interleukin 10 (IL-10) and IL-12 was analyzed in the salivary gland tissues of MRL/lpr mice with Sjögren's syndrome. We demonstrate a significant role of IL-10 and IL-12 in vivo during development of Sjögren's syndrome in MRL/lpr mice. IL-10 mRNA expression was detected before the onset of disease and was upregulated during the course of autoimmune sialadenitis by RT-PCR. A predominant level of expression of IL-12 mRNA was also detected earlier in the proinflammatory stage of autoimmune sialadenities. Moreover, MHC class II (I-Ak) mRNA was detected before the onset of inflammatory lesions until older ages in the salivary glands of MRL/lpr mice. These results suggest that endogenous IL-10 and IL-12 may play important roles on immune-mediated destruction of the salivary glands during development of organ-specific autoimmunity in MRL/lpr mice.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autoimmune Diseases / genetics
  • Autoimmune Diseases / physiopathology*
  • Base Sequence
  • Disease Models, Animal
  • Disease Progression
  • Female
  • Gene Expression Regulation*
  • Histocompatibility Antigens Class II / biosynthesis
  • Histocompatibility Antigens Class II / genetics
  • Interleukin-10 / biosynthesis
  • Interleukin-10 / genetics
  • Interleukin-10 / physiology*
  • Interleukin-12 / biosynthesis
  • Interleukin-12 / genetics
  • Interleukin-12 / physiology*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Mutant Strains
  • Molecular Sequence Data
  • Organ Specificity
  • Polymerase Chain Reaction
  • RNA, Messenger
  • Salivary Glands / metabolism*
  • Salivary Glands / pathology
  • Sialadenitis / etiology
  • Sialadenitis / genetics
  • Sialadenitis / physiopathology
  • Sjogren's Syndrome / genetics
  • Sjogren's Syndrome / physiopathology*
  • Spleen / metabolism
  • Spleen / pathology
  • fas Receptor / genetics
  • fas Receptor / physiology*

Substances

  • Histocompatibility Antigens Class II
  • RNA, Messenger
  • fas Receptor
  • Interleukin-10
  • Interleukin-12