Fibrinolytic abnormalities in two different cutaneous manifestations of venous disease

J Am Acad Dermatol. 1996 Feb;34(2 Pt 1):204-8. doi: 10.1016/s0190-9622(96)80113-7.

Abstract

Background: Chronic venous insufficiency may be associated with lipodermatosclerosis or atrophie blanche. Coagulation abnormalities may be related to these cutaneous disorders.

Objective: Our purpose was to determine whether fibrinolytic abnormalities exist in patients with lipodermatosclerosis or atrophie blanche.

Methods: A case control study of patients with venous disease and atrophie blanche or lipodermatosclerosis was performed. Plasma levels of tissue plasminogen activator (t-PA) and plasminogen activator inhibitor-1 (PAI-1) in a resting and venous occluded state were measured.

Results: Plasma levels of PAI-1 were different between the two groups of patients. The lipodermatosclerosis group had significantly higher levels of PAI-1 in both the resting and venous occluded states (p < 0.001). Patients with atrophie blanche had milder elevations of PAI-1 in the resting and venous occluded state (p = 0.06).

Conclusion: Fibrinolytic abnormalities are present in patients with venous disease. These abnormalities are different between patients with lipodermatosclerosis and patients with atrophie blanche.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Aged
  • Analysis of Variance
  • Atrophy
  • Blood Coagulation Disorders / physiopathology
  • Case-Control Studies
  • Female
  • Fibrinolysis* / physiology
  • Humans
  • Male
  • Middle Aged
  • Plasminogen Activator Inhibitor 1 / blood
  • Rest
  • Scleroderma, Localized / blood
  • Scleroderma, Localized / physiopathology*
  • Skin / pathology*
  • Skin Diseases, Vascular / blood
  • Skin Diseases, Vascular / physiopathology*
  • Telangiectasis / blood
  • Telangiectasis / physiopathology
  • Thrombophlebitis / blood
  • Thrombophlebitis / physiopathology
  • Tissue Plasminogen Activator / blood
  • Varicose Ulcer / blood
  • Varicose Ulcer / physiopathology
  • Venous Insufficiency / blood
  • Venous Insufficiency / physiopathology*

Substances

  • Plasminogen Activator Inhibitor 1
  • Tissue Plasminogen Activator