Pycnodysostosis, a lysosomal disease caused by cathepsin K deficiency

Science. 1996 Aug 30;273(5279):1236-8. doi: 10.1126/science.273.5279.1236.

Abstract

Pycnodysostosis, an autosomal recessive osteochondrodysplasia characterized by osteosclerosis and short stature, maps to chromosome 1q21. Cathepsin K, a cysteine protease gene that is highly expressed in osteoclasts, localized to the pycnodysostosis region. Nonsense, missense, and stop codon mutations in the gene encoding cathepsin K were identified in patients. Transient expression of complementary DNA containing the stop codon mutation resulted in messenger RNA but no immunologically detectable protein. Thus, pycnodysostosis results from gene defects in a lysosomal protease with highest expression in osteoclasts. These findings suggest that cathepsin K is a major protease in bone resorption, providing a possible rationale for the treatment of disorders such as osteoporosis and certain forms of arthritis.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Base Sequence
  • Bone Matrix / metabolism
  • Bone Resorption
  • Cathepsin K
  • Cathepsins / deficiency
  • Cathepsins / genetics*
  • Cathepsins / metabolism
  • Chromosome Mapping
  • Chromosomes, Human, Pair 1
  • Codon, Terminator
  • Dinucleoside Phosphates / genetics
  • Humans
  • Lysosomal Storage Diseases / enzymology
  • Lysosomal Storage Diseases / genetics*
  • Lysosomes / enzymology*
  • Molecular Sequence Data
  • Mutagenesis, Site-Directed
  • Mutation
  • Osteochondrodysplasias / enzymology
  • Osteochondrodysplasias / genetics*
  • Osteoclasts / enzymology*
  • Transfection

Substances

  • Codon, Terminator
  • Dinucleoside Phosphates
  • cytidylyl-3'-5'-guanosine
  • Cathepsins
  • cathepsin S
  • CTSK protein, human
  • Cathepsin K