Saturable binding sites for the coenzyme A ester of nafenopin, a peroxisome proliferator, in rat liver cytosol

Xenobiotica. 1995 Dec;25(12):1293-300. doi: 10.3109/00498259509061918.

Abstract

1. At least three different molecular weight binding sites exist in rat liver cytosol for nafenopin-CoA, the coenzyme A ester and metabolic product of the carcinogenic peroxisome proliferator nafenopin. No binding sites for the free drug were observed. 2. Polypeptides of 35-40 kDa molecular weight range where no acyl-CoA binding proteins have been previously described bind the highest proportion of nafenopin-CoA (60-70%). Binding is displaceable by the CoA esters of other peroxisome proliferators (ciprofibrate and tibric acid) and also by oleoyl-CoA but by palmitoyl-CoA. Direct binding studies show that 35-40-kDa polypeptides bind oleoyl-CoA but not oleic or palmitic acid, or palmitoyl-CoA. 3. Polypeptides of 10-14 and 65-70 kDa also bind nafenopin-CoA. However, in contrast with 35-40-kDa polypeptides they also bind oleic and palmitic acid as well as their correspondent acyl-CoA thioesters.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetyl Coenzyme A / metabolism
  • Acyl Coenzyme A / metabolism*
  • Animals
  • Binding Sites
  • Chromatography, Gel
  • Cytosol / metabolism
  • Hypolipidemic Agents / metabolism
  • Hypolipidemic Agents / pharmacology
  • Kinetics
  • Liver / metabolism*
  • Male
  • Microbodies / drug effects*
  • Microbodies / metabolism
  • Molecular Weight
  • Nafenopin / analogs & derivatives*
  • Nafenopin / metabolism
  • Nafenopin / pharmacology
  • Protein Binding
  • Proteins / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Tritium

Substances

  • Acyl Coenzyme A
  • Hypolipidemic Agents
  • Proteins
  • Nafenopin
  • Tritium
  • nafenopin-coenzyme A
  • Acetyl Coenzyme A