Ionizing radiation-induced apoptosis is associated with c-Jun expression and c-Jun/AP-1 activation in the developing cerebellum of the rat

Neurosci Lett. 1995 Dec 29;202(1-2):105-8. doi: 10.1016/0304-3940(95)12220-6.

Abstract

Previous studies have shown that ionizing radiation-induced cell death in the developing brain has morphological characteristics of apoptosis and is associated with internucleosomal DNA fragmentation. In the present study, we have observed c-Jun induction in cells sensitive to ionizing radiation during the whole process of radiation-induced cell death, and that this expression is accompanied by modifications in the composition of AP-1 complexes: c-Jun/AP-1 activity is highly increased whereas Jun D/AP-1 is slightly decreased. These results show that c-Jun expression and c-Jun/AP-1 activity are induced in the developing brain following ionizing radiation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibody Specificity
  • Apoptosis / genetics*
  • Apoptosis / radiation effects*
  • Base Sequence
  • Cerebellum / cytology
  • Cerebellum / embryology
  • Cerebellum / physiology*
  • DNA / analysis
  • Immunohistochemistry
  • Molecular Sequence Data
  • Proto-Oncogene Proteins c-jun / genetics
  • Proto-Oncogene Proteins c-jun / immunology
  • Proto-Oncogene Proteins c-jun / radiation effects*
  • Rats
  • Rats, Sprague-Dawley
  • Transcription Factor AP-1 / genetics
  • Transcription Factor AP-1 / radiation effects

Substances

  • Proto-Oncogene Proteins c-jun
  • Transcription Factor AP-1
  • DNA