Rapid induction of tumor necrosis factor cytotoxicity in naive splenic T cells by simultaneous CD80 (B7.1) and CD54 (ICAM-1) co-stimulation

Eur J Immunol. 1996 Sep;26(9):2160-4. doi: 10.1002/eji.1830260930.

Abstract

The co-expression of B7.1 (CD80) and intercellular adhesion molecule (ICAM)-1 (CD54) on tumor cells can induce tumor immunity and immunological memory. We show here that the non-immunogenic tumor lines Lewis lung carcinoma and B16F10 melanoma, co-transfected with B7.1 and ICAM-1, induced cytotoxic levels of membrane tumor necrosis factor (TNF) on naive syngeneic T cells within 24 h. Membrane TNF expression, primarily on CD4 cells, was responsible for tumor cell lysis by naive spleen cells and could be completely abolished by anti-TNF antiserum. It is suggested that the strong induction of TNF cytotoxicity may be important in the establishment of tumor immunity.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • B7-1 Antigen / physiology*
  • Cell Aggregation
  • Cricetinae
  • Cytotoxicity, Immunologic*
  • Female
  • Intercellular Adhesion Molecule-1 / physiology*
  • Mice
  • Mice, Inbred C57BL
  • Spleen / immunology
  • T-Lymphocytes / immunology*
  • Tumor Necrosis Factor-alpha / physiology*

Substances

  • B7-1 Antigen
  • Tumor Necrosis Factor-alpha
  • Intercellular Adhesion Molecule-1