Autoradiographic localization of vasopressin V1a receptors in the rat kidney using [3H]-SR 49059

Kidney Int. 1996 Aug;50(2):499-505. doi: 10.1038/ki.1996.341.

Abstract

Localization and characterization of binding sites of the selective non-peptide vasopressin receptor V1a ligand, [3H]-SR 49059, were investigated in the adult rat kidney by quantitative autoradiography using a fast-detecting radioluminographic phosphor-imaging plate system. [3H]-SR 49059, like the other V1a ligands used, showed a total absence of binding in the papilla, discrete and sparse labeling in the cortex and maximal binding in the outer part of the inner medulla. This labeling seemed to be mainly associated with medullary interstitial cells and vascular elements of the vasa recta. Conversely, [3H]-AVP intensely labeled the V2-enriched medulla-papillary portion of the kidney and, to a lesser extent, the cortical structures. [3H]-SR 49059 binding, quantified in the outer part of the inner medulla in rat kidney sections, was time-dependent, reversible, saturable and a single class of high affinity binding sites (Kd = 1.48 +/- 0.16 nM) was identified. The relative potencies of the reference peptide and non-peptide compounds to inhibit [3H]-SR 49059 binding confirm the V1a nature of the site and the stereospecificity of this binding. Thus, [3H]-SR 49059 allows the mapping and characterization of the V1a receptor population present in the rat kidney. The stability and the highly selective affinity of this non-peptide ligand for rat and human V1a receptors make it a suitable probe for the localization of V1a receptors in organs expressing heterogeneous populations of receptors.

MeSH terms

  • Animals
  • Arginine Vasopressin / antagonists & inhibitors
  • Arginine Vasopressin / metabolism
  • Autoradiography
  • Hormone Antagonists / metabolism
  • Humans
  • Indoles / metabolism*
  • Kidney / metabolism*
  • Kinetics
  • Liver / metabolism
  • Male
  • Pyrrolidines / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Vasopressin / metabolism*
  • Tissue Distribution
  • Tritium

Substances

  • Hormone Antagonists
  • Indoles
  • Pyrrolidines
  • Receptors, Vasopressin
  • Tritium
  • Arginine Vasopressin
  • relcovaptan