Monocyte-derived macrophages prime peripheral T cells to undergo apoptosis by cell-cell contact via ICAM-1/LFA-1-dependent mechanism

Immunobiology. 1996 Aug;195(3):323-33. doi: 10.1016/S0171-2985(96)80049-0.

Abstract

We designed the present study to clarify the mechanism of superantigen-induced apoptosis of human mature T cells and to elucidate the pivotal roles of monocyte-derived macrophages in induction of T cell apoptosis. Exposure of unfractionated human peripheral blood mononuclear cells to SEA, SEB or PHA elicited apoptosis in T cells after 5-day culture. In purified T cell preparations, SEB was unable to induce apoptosis, but was inductive when the purified T cells were cocultured with monocyte-derived macrophages adhering to plastic culture dishes. Placing the T cells in the insert wells which physically separated them from the adhering macrophages resulted in a complete loss of SEB-induced apoptosis. The addition of blocking antibodies against LFA-1, ICAM-1 and CD2 to the cocultures significantly inhibited the SEB-induced T cell apoptosis. We concluded therefore that direct contact of macrophages with T cells is critical in SEB-induced T cell apoptosis, and that adhesion molecules such as LFA-1/ICAM-1 and CD2 may be involved in the mechanism of this effect.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects*
  • Apoptosis / immunology*
  • CD2 Antigens / pharmacology
  • Cell Adhesion / immunology
  • Cells, Cultured
  • Humans
  • Immunologic Memory / immunology*
  • Intercellular Adhesion Molecule-1 / pharmacology*
  • Ligands
  • Lymphocyte Activation / immunology
  • Lymphocyte Function-Associated Antigen-1 / pharmacology*
  • Macrophages / cytology
  • Macrophages / physiology*
  • Superantigens / immunology
  • Superantigens / pharmacology
  • T-Lymphocytes / immunology*

Substances

  • CD2 Antigens
  • Ligands
  • Lymphocyte Function-Associated Antigen-1
  • Superantigens
  • Intercellular Adhesion Molecule-1