Abstract
In human neocortical slices the specific L-type calcium channel blocker verapamil had been shown to be antiepileptic in the low Mg(2+)-model of epilepsy. The present investigation demonstrated: (1) verapamil exerted also an antiepileptic effect on epileptiform field potentials (EFP) induced by the GABAA-antagonist bicuculline. (2) The unspecific calcium channel modulator flunarizine, which in contrast to verapamil penetrates the blood-brain barrier, depressed EFP in the low Mg(2+)-model and in the bicuculline model. (3) There was no significant difference in the antiepileptic efficacy of verapamil and flunarizine in epileptic (epilepsy surgery) and primary non-epileptic (tumor surgery) neocortical slices.
MeSH terms
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Astrocytoma / metabolism
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Astrocytoma / surgery
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Bicuculline / pharmacology*
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Brain Neoplasms / metabolism
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Brain Neoplasms / physiopathology
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Brain Neoplasms / secondary
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Brain Neoplasms / surgery
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Cerebral Cortex / drug effects
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Cerebral Cortex / metabolism
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Cerebral Cortex / physiopathology*
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Ependymoma / metabolism
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Ependymoma / physiopathology
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Epilepsy, Frontal Lobe / metabolism*
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Epilepsy, Frontal Lobe / physiopathology
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Epilepsy, Temporal Lobe / metabolism
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Epilepsy, Temporal Lobe / physiopathology*
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Epilepsy, Temporal Lobe / surgery
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Evoked Potentials / drug effects
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Female
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Flunarizine / pharmacology*
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Humans
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In Vitro Techniques
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Magnesium / pharmacology*
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Male
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Oligodendroglioma / metabolism
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Oligodendroglioma / physiopathology
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Verapamil / pharmacology*
Substances
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Verapamil
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Magnesium
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Flunarizine
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Bicuculline