The carboxyl termini of beta-amyloid peptides 1-40 and 1-42 are generated by distinct gamma-secretase activities

J Biol Chem. 1996 Nov 8;271(45):28655-9. doi: 10.1074/jbc.271.45.28655.

Abstract

We have studied the effects of peptide aldehyde protease inhibitors on the secretion of beta-amyloid peptide 1-40 (Abeta(1-40)) and Abeta(1-42) by HEK 293 and COS-1 cells expressing beta-amyloid precursor protein with the Swedish double mutation. A multiphasic SDS-polyacrylamide gel electrophoresis system was used for the discrimination of Abeta(1-40) and Abeta(1-42). Calpain inhibitor I, carbobenzoxyl-Leu-Leu-leucinal, and calpeptin were found to reduce the amount of Abeta(1-40) released into the medium in a dose-dependent manner. The reduction of Abeta(1-40) after treatment with 50 microM calpain inhibitor I or 5 microM carbobenzoxyl-Leu-Leu-leucinal was accompanied by a slight increase of Abeta(1-42) released into the medium. These observations suggest that the cleavages at residues 40 and 42 are accomplished by different enzyme activities.

MeSH terms

  • Amyloid Precursor Protein Secretases
  • Amyloid beta-Peptides / metabolism*
  • Animals
  • Aspartic Acid Endopeptidases
  • COS Cells
  • Cysteine Proteinase Inhibitors / metabolism
  • Electrophoresis, Polyacrylamide Gel
  • Endopeptidases / metabolism*
  • Glycoproteins / metabolism
  • Humans
  • Leupeptins / metabolism
  • Peptide Fragments / metabolism*
  • Tumor Cells, Cultured

Substances

  • Amyloid beta-Peptides
  • Cysteine Proteinase Inhibitors
  • Glycoproteins
  • Leupeptins
  • Peptide Fragments
  • amyloid beta-protein (1-40)
  • amyloid beta-protein (1-42)
  • calpain inhibitors
  • Amyloid Precursor Protein Secretases
  • Endopeptidases
  • Aspartic Acid Endopeptidases
  • BACE1 protein, human
  • benzyloxycarbonylleucyl-leucyl-leucine aldehyde