Ionic channels in corneal endothelium

Am J Physiol. 1996 Apr;270(4 Pt 1):C975-89. doi: 10.1152/ajpcell.1996.270.4.C975.

Abstract

Single-channel patch-clamp techniques as well as standard and perforated-patch whole cell voltage-clamp techniques have been applied to the study of ionic channels in the corneal endothelium of several species. These studies have revealed two major K+ currents. One is due to an anion- and temperature-stimulated channel that is blocked by Cs+ but not by most other K+ channel blockers, and the other is similar to the family of A-currents found in excitable cells. The A-current is transient after a depolarizing voltage step and is blocked by both 4-aminopyridine and quinidine. These two currents are probably responsible for setting the -50 to -60 mV resting voltage reported for these cells. A Ca(2+)-activated ATP-inhibited nonselective cation channel and a tetrodotoxin-blocked Na+ channel are possible Na+ inflow pathways, but, given their gating properties, it is not certain that either channel works under physiological conditions. A large-conductance anion channel has also been identified by single-channel patch-clamp techniques. Single corneal endothelial cells have input resistances of 5-10 G omega and have steady-state K+ currents that are approximately 10 pA at the resting voltage. Pairs or monolayers of cells are electrically coupled and dye coupled through gap junctions.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Chloride Channels / physiology
  • Electrophysiology
  • Endothelium, Corneal / metabolism*
  • Gap Junctions / physiology
  • Humans
  • Ion Channels / physiology*
  • Potassium Channels / physiology
  • Sodium Channels / physiology

Substances

  • Chloride Channels
  • Ion Channels
  • Potassium Channels
  • Sodium Channels