Abstract
Prostacyclin, one of the major prostanoids generated in adipose tissue, has been previously described as an autocrine/paracrine adipogenic effector, acting, in preadipose cells, by means of cAMP and free Ca2+ as cell surface receptor-mediated messengers. The present study presents evidence for the first time that its stable analogue, carbaprostacyclin, is unique among prostanoids in regulating the expression of two differentiation-dependent genes in preadipose and adipose cells in a way distinct from that elicited by its cell surface receptor. This regulation is likely mediated by some member(s) of the peroxisome proliferator-activated receptor family and suggests that prostacyclin behaves as an intracrine effector of adipose cell differentiation.
Publication types
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Comparative Study
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Research Support, Non-U.S. Gov't
MeSH terms
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3T3 Cells
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Adipocytes / cytology
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Adipocytes / drug effects
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Adipocytes / metabolism*
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Angiotensinogen / biosynthesis
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Angiotensinogen / genetics*
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Animals
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Carrier Proteins / biosynthesis
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Carrier Proteins / genetics*
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Cell Differentiation
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Cell Line
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Dexamethasone / pharmacology
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Dose-Response Relationship, Drug
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Epoprostenol / analogs & derivatives*
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Epoprostenol / pharmacology
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Fatty Acid-Binding Protein 7
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Fatty Acid-Binding Proteins
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Gene Expression Regulation / drug effects*
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Hormone Antagonists / pharmacology
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Mice
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Mifepristone / pharmacology
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Myelin P2 Protein / biosynthesis
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Myelin P2 Protein / genetics*
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Neoplasm Proteins*
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Nerve Tissue Proteins*
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Prostaglandins / pharmacology
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RNA, Messenger / genetics
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RNA, Messenger / metabolism
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Receptors, Epoprostenol
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Receptors, Prostaglandin / metabolism
Substances
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Carrier Proteins
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Fabp5 protein, mouse
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Fabp7 protein, mouse
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Fatty Acid-Binding Protein 7
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Fatty Acid-Binding Proteins
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Hormone Antagonists
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Myelin P2 Protein
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Neoplasm Proteins
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Nerve Tissue Proteins
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Prostaglandins
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RNA, Messenger
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Receptors, Epoprostenol
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Receptors, Prostaglandin
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Angiotensinogen
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Mifepristone
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carboprostacyclin
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Dexamethasone
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Epoprostenol