Endothelin antagonist bosentan blocks neurogenic inflammation, but is not effective in aborting migraine attacks

Pain. 1996 Oct;67(2-3):375-8. doi: 10.1016/0304-3959(96)03137-5.

Abstract

Bosentan, a specific mixed antagonist of endothelin receptors with no vasoconstrictor activity, inhibits neurogenic plasma extravasation (NPE) within rat dura mater. This would predict efficacy in aborting migraine attacks, without causing cardiovascular side-effects. We investigated the efficacy of 250 mg i.v. bosentan in a randomized, double-blind, placebo-controlled, clinical trial. Improvement from moderate/severe to mild/no headache at 2 h (primary efficacy measure) occurred in 5/23 (22%) of bosentan-treated and in 9/25 (36%) of placebo-treated patients (effect difference -14%; 95% CI -52%, 24%). Thus, inhibition of NPE may not predict clinical efficacy of experimental antimigraine drugs. Vasoconstrictor action may be needed.

Publication types

  • Clinical Trial
  • Multicenter Study
  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Blood Flow Velocity / drug effects
  • Bosentan
  • Cerebrovascular Circulation / drug effects
  • Double-Blind Method
  • Dura Mater / drug effects*
  • Endothelins / antagonists & inhibitors*
  • Female
  • Humans
  • Inflammation / prevention & control
  • Male
  • Middle Aged
  • Sulfonamides / therapeutic use*
  • Treatment Failure

Substances

  • Endothelins
  • Sulfonamides
  • Bosentan