p27KIP1 blocks cyclin E-dependent transactivation of cyclin A gene expression

Mol Cell Biol. 1997 Jan;17(1):407-15. doi: 10.1128/MCB.17.1.407.

Abstract

Cyclin E is necessary and rate limiting for the passage of mammalian cells through the G1 phase of the cell cycle. Control of cell cycle progression by cyclin E involves cdk2 kinase, which requires cyclin E for catalytic activity. Expression of cyclin E/cdk2 leads to an activation of cyclin A gene expression, as monitored by reporter gene constructs derived from the human cyclin A promoter. Promoter activation by cyclin E/cdk2 requires an E2F binding site in the cyclin A promoter. We show here that cyclin E/cdk2 kinase can directly bind to E2F/p107 complexes formed on the cyclin A promoter-derived E2F binding site, and this association is controlled by p27KIP1, most likely through direct protein-protein interaction. These observation suggest that cyclin E/cdk2 associates with E2F/p107 complexes in late G1 phase, once p27KIP1 has decreased below a critical threshold level. Since a kinase-negative mutant of cdk2 prevents promoter activation, it appears that transcriptional activation of the cyclin A gene requires an active cdk2 kinase tethered to its promoter region.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3 Cells
  • Animals
  • Binding Sites
  • CDC2-CDC28 Kinases*
  • Carrier Proteins*
  • Cell Cycle Proteins*
  • Cyclin-Dependent Kinase 2
  • Cyclin-Dependent Kinase Inhibitor p27
  • Cyclin-Dependent Kinases / antagonists & inhibitors
  • Cyclin-Dependent Kinases / genetics
  • Cyclin-Dependent Kinases / metabolism
  • Cyclins / genetics*
  • Cyclins / metabolism*
  • DNA-Binding Proteins*
  • E2F Transcription Factors
  • Enzyme Inhibitors
  • G1 Phase / physiology
  • Humans
  • Mice
  • Microtubule-Associated Proteins / analysis
  • Microtubule-Associated Proteins / physiology*
  • Models, Genetic
  • Nuclear Proteins / genetics
  • Nuclear Proteins / metabolism
  • Promoter Regions, Genetic / genetics
  • Protein Binding
  • Protein Serine-Threonine Kinases / antagonists & inhibitors
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / metabolism
  • Recombinant Fusion Proteins
  • Retinoblastoma-Binding Protein 1
  • Retinoblastoma-Like Protein p107
  • Transcription Factor DP1
  • Transcription Factors / metabolism
  • Transcriptional Activation / physiology*
  • Transfection
  • Tumor Suppressor Proteins*

Substances

  • Arid4a protein, mouse
  • Carrier Proteins
  • Cdkn1b protein, mouse
  • Cell Cycle Proteins
  • Cyclins
  • DNA-Binding Proteins
  • E2F Transcription Factors
  • Enzyme Inhibitors
  • Microtubule-Associated Proteins
  • Nuclear Proteins
  • RBL1 protein, human
  • Rbl1 protein, mouse
  • Recombinant Fusion Proteins
  • Retinoblastoma-Binding Protein 1
  • Retinoblastoma-Like Protein p107
  • Transcription Factor DP1
  • Transcription Factors
  • Tumor Suppressor Proteins
  • Cyclin-Dependent Kinase Inhibitor p27
  • Protein Serine-Threonine Kinases
  • CDC2-CDC28 Kinases
  • CDK2 protein, human
  • Cdk2 protein, mouse
  • Cyclin-Dependent Kinase 2
  • Cyclin-Dependent Kinases