Polymorphisms within the human tumor necrosis factor-alpha promoter region in human immunodeficiency virus type 1-seropositive persons

J Infect Dis. 1997 Jan;175(1):188-90. doi: 10.1093/infdis/175.1.188.

Abstract

The question is addressed whether particular tumor necrosis factor-alpha (TNF-alpha) polymorphisms are associated with clinical course and outcome of human immunodeficiency virus type 1 (HIV-1) infection. The distribution of four TNF-alpha guanine (G) to adenosine (A) transition polymorphisms at positions -376, -308, -238, and -163 of the 5' promoter region of the TNF-alpha gene was studied in a nested case-control study among HIV-1-seropositive participants of the Amsterdam Cohort. None of the polymorphisms was significantly associated with long-term asymptomatic survival after HIV-1 infection compared with progression to clinical AIDS. Moreover, specific AIDS-defining illnesses or biologic phenotype of the HIV-1 virus were not associated with TNF-alpha alleles. The results of this study do not point toward a role for known TNF-alpha G to A transition polymorphisms in the clinical course of HIV-1 infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acquired Immunodeficiency Syndrome / genetics
  • Acquired Immunodeficiency Syndrome / physiopathology
  • Acquired Immunodeficiency Syndrome / virology
  • Alleles
  • Case-Control Studies
  • Cytopathogenic Effect, Viral
  • Disease Progression
  • Giant Cells
  • HIV Infections / genetics*
  • HIV Infections / physiopathology
  • HIV Infections / virology
  • HIV Seropositivity / genetics
  • HIV Seropositivity / physiopathology
  • HIV Seropositivity / virology
  • HIV-1* / pathogenicity
  • Humans
  • Polymorphism, Genetic*
  • Promoter Regions, Genetic / genetics*
  • Tumor Necrosis Factor-alpha / genetics*

Substances

  • Tumor Necrosis Factor-alpha