Lactoferrin as a suppressor of cell migration of gastrointestinal cell lines

J Cell Physiol. 1997 Feb;170(2):101-5. doi: 10.1002/(SICI)1097-4652(199702)170:2<101::AID-JCP1>3.0.CO;2-S.

Abstract

The effects of lactoferrin (Lf), an iron-binding glycoprotein, on cell migration were investigated. Lf inhibited the cell migration of three gastrointestinal cell lines (Caco-2 cells, AGS cells, and IEC-18 cells) in vitro. Both iron-saturated (holo) and iron-depleted (apo) Lf showed this inhibitory effect. Chelation of iron in the culture medium by desferrioxamine did not affect the activity of either form of Lf. A pepsin hydrolysate of Lf exhibited effectiveness similar to that of intact Lf. These results demonstrate a novel activity of Lf and suggest a potential role for this molecule in gastrointestinal wound healing, which is independent of its iron-binding capacity.

MeSH terms

  • Adenocarcinoma
  • Animals
  • Apoproteins / pharmacology
  • Cattle
  • Cell Movement / drug effects*
  • Colonic Neoplasms
  • Deferoxamine / pharmacology
  • Digestive System Physiological Phenomena*
  • Humans
  • Intestinal Mucosa
  • Kinetics
  • Lactoferrin / pharmacology*
  • Rats
  • Stomach Neoplasms

Substances

  • Apoproteins
  • apolactoferrin
  • Lactoferrin
  • Deferoxamine