Devazepide (L-364718) inhibits growth and increases expression of tumor markers in HT29-S-B6 cells

C R Acad Sci III. 1996 Dec;319(12):1133-8.

Abstract

Devazepide (L-364718, a non-peptide antagonist of CCK-A receptors), inhibits the proliferation and induces morphologic changes in the mucous-secreting, autonomously proliferating human cancer colon cell line (HT29-S-B6. Addition of devazepide (10 microM) for at least 3 days in the exponential phase of growth enhanced the baseline production of gastric M1 mucins 2-3-fold and that of carcinoembryonic antigens 5-fold. Moreover, devazepide induced an increase in the amount of the MUC-5AC mRNA expressed by HT29-S-B6 cells. The increased in mucins secretion induced by devazepide was persistent after removal and independent of the presence of serum. In conclusion, devazepide-L-364718 behaves as a maturation agent in the cell clone HT29-S-B6.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Benzodiazepinones / pharmacology*
  • Carcinoembryonic Antigen / analysis*
  • Cell Division / drug effects
  • Clone Cells / cytology
  • Clone Cells / metabolism
  • Devazepide
  • Gastric Mucins / analysis*
  • Gastric Mucins / metabolism
  • HT29 Cells / cytology*
  • HT29 Cells / metabolism
  • Hormone Antagonists / pharmacology*
  • Humans
  • Receptors, Cholecystokinin / antagonists & inhibitors*
  • Tumor Cells, Cultured

Substances

  • Benzodiazepinones
  • Carcinoembryonic Antigen
  • Gastric Mucins
  • Hormone Antagonists
  • Receptors, Cholecystokinin
  • Devazepide