Myosin as cofactor and substrate in fibrinolysis

FEBS Lett. 1997 Apr 21;407(1):93-6. doi: 10.1016/s0014-5793(97)00303-7.

Abstract

Myosin accelerates plasminogen activation by tissue-type plasminogen activator (tPA), and is degraded extensively by plasmin. Myosin binds both tPA and plasminogen, and enhances activation of des1-77-plasminogen by tPA but not by urokinase-type plasminogen activator (uPA). Myosin decreases K(M) and increases k(cat) for des1-77-plasminogen activation by tPA, to yield catalytic efficiencies in excess of 8000 M-1 s-1. The effect of myosin is attributed to its C-terminal portion, the myosin rod. With a K(M) of 3 microM, myosin is a high-affinity substrate for plasmin. The findings indicate that myosin is a cofactor for plasminogen activation and a substrate for plasmin.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Enzyme Activation
  • Fibrinolysin / metabolism*
  • Fibrinolysis / physiology*
  • Humans
  • Myosins / metabolism*
  • Peptide Fragments / metabolism
  • Plasminogen / metabolism*
  • Tissue Plasminogen Activator / metabolism*
  • Urokinase-Type Plasminogen Activator / metabolism

Substances

  • Peptide Fragments
  • Plasminogen
  • Tissue Plasminogen Activator
  • Fibrinolysin
  • Urokinase-Type Plasminogen Activator
  • Myosins