Polymorphism of the HLA-DMA and DMB genes in rheumatoid arthritis

Arthritis Rheum. 1997 May;40(5):854-8. doi: 10.1002/art.1780400512.

Abstract

Objective: To determine whether the HLA-DMA and DMB genes, whose encoded molecules are involved in HLA class II-restricted antigen presentation, contribute to the genetic susceptibility to rheumatoid arthritis (RA).

Methods: One hundred ninety-one RA patients, 147 control subjects, and 218 HLA-DRB1 genotype-matched control subjects were oligotyped for DMA and DMB genes.

Results: DMA*0103 and DMB*0104 were significantly increased in the RA patients compared with the randomly selected and the matched controls, thus indicating a direct influence of the DM genes. After stratification of the patients and matched controls according to DRB1 genotypes, only DMA*0103 was increased in the RA patients with DRB1*01, as well as in the patients negative for the RA-susceptibility DR alleles.

Conclusion: Our results suggest that DMA*0103 could play an additional role in the genetic susceptibility to RA.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alleles
  • Arthritis, Rheumatoid / genetics*
  • Genetic Predisposition to Disease
  • HLA-D Antigens / genetics*
  • Histocompatibility Antigens Class II*
  • Humans
  • Polymorphism, Genetic

Substances

  • H2-M antigens
  • HLA-D Antigens
  • HLA-DM antigens
  • Histocompatibility Antigens Class II