Absence of RET proto-oncogene mutations in a father and son with pheochromocytoma and pancreatic islet cell tumor

Int J Urol. 1997 Mar;4(2):169-71. doi: 10.1111/j.1442-2042.1997.tb00165.x.

Abstract

Background: We describe a father and son with a combination of pheochromocytoma and pancreatic islet cell tumor. Although its familial occurrence is rare, this syndrome could be called overlapping-type multiple endocrine neoplasia (MEN), since it fulfills the criteria for both type 1 and type 2 MEN. Recently, germ line mutations of the RET proto-oncogene (RET) were found to be related to tumorigenesis and disease phenotypes in type 2 MEN.

Methods: Using polymerase chain reaction-single strand conformation polymorphism (PCR-SSCP) analysis, we looked for germ line mutations of RET in 8 members of this family, including the 2 patients.

Results: Analysis of RET exons 10, 11 and 16, which contain the hot-spot codons for MEN type 2, revealed no mutations in any individual examined.

Conclusion: These findings suggest that these 3 exons in RET are not related to tumorigenesis in overlapping-type MEN.

Publication types

  • Case Reports

MeSH terms

  • Adenoma, Islet Cell / genetics*
  • Adolescent
  • Adrenal Gland Neoplasms / genetics*
  • Adult
  • Drosophila Proteins*
  • Humans
  • Male
  • Mutation*
  • Neoplasms, Multiple Primary
  • Pancreatic Neoplasms / genetics*
  • Pheochromocytoma / genetics*
  • Polymerase Chain Reaction
  • Polymorphism, Single-Stranded Conformational
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins c-ret
  • Proto-Oncogenes*
  • Receptor Protein-Tyrosine Kinases / genetics*

Substances

  • Drosophila Proteins
  • MAS1 protein, human
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-ret
  • Receptor Protein-Tyrosine Kinases
  • Ret protein, Drosophila