Cloning and characterization of a novel member of the human Mad gene family (MADH6)

Genomics. 1997 Jun 15;42(3):446-51. doi: 10.1006/geno.1997.4753.

Abstract

MAD (mothers against decapentaplegic)-related proteins (MADRs) are intracellular components that play critical roles in signal-transduction pathways involving the transforming growth factor beta (TGFbeta) superfamily. Some Mad genes are candidates for tumor-suppressor functions. From a human fetal brain cDNA library we have isolated a novel Mad-related gene. Two alternatively transcribed mRNAs encode deduced 430- and 467-amino-acid peptides that showed high levels of similarity to MADR1/Smad1/hMAD1 (about 80% identity at the amino acid level). This gene, which we designated MADH6, resides on 13q12-q14 between BRCA2 and RB, a region that frequently displays loss of heterozygosity in breast, liver, and prostate cancers.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Base Sequence
  • Chromosomes, Human, Pair 13*
  • Cloning, Molecular
  • DNA, Complementary
  • Gene Expression
  • Humans
  • Molecular Sequence Data
  • Peptides
  • Proteins / genetics*
  • Sequence Analysis, DNA
  • Sequence Homology, Amino Acid
  • Smad8 Protein
  • Tissue Distribution
  • Transcription Factors*
  • Transforming Growth Factor beta / genetics*

Substances

  • DNA, Complementary
  • Peptides
  • Proteins
  • SMAD9 protein, human
  • Smad8 Protein
  • Transcription Factors
  • Transforming Growth Factor beta

Associated data

  • GENBANK/D83760
  • GENBANK/D83761