Interaction of the UV-damaged DNA-binding protein with hepatitis B virus X protein is conserved among mammalian hepadnaviruses and restricted to transactivation-proficient X-insertion mutants

J Virol. 1997 Aug;71(8):6194-9. doi: 10.1128/JVI.71.8.6194-6199.1997.

Abstract

We carried out a comparative analysis of several proposed host protein partners of the human hepatitis B virus X protein (HBx) using both the GAL4- and the LexA-based yeast two-hybrid system. We showed that the interaction of HBx with the UV-damaged DNA-binding protein (UVDDB) is positive in both yeast systems, detectable in cotransfected human cells, conserved by rodent hepadnavirus X proteins (known to transactivate in human cells), and tightly correlated with the transactivation proficiency of X-insertion mutants. Taken together, our results strongly suggest that UVDDB is involved in X-mediated transactivation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Binding Sites
  • DNA-Binding Proteins / metabolism*
  • DNA-Binding Proteins / radiation effects
  • Hepadnaviridae / genetics*
  • Humans
  • Mutation
  • Trans-Activators / physiology*
  • Transcriptional Activation*
  • Ultraviolet Rays
  • Viral Regulatory and Accessory Proteins

Substances

  • DNA-Binding Proteins
  • Trans-Activators
  • Viral Regulatory and Accessory Proteins
  • hepatitis B virus X protein