Malignant lymphoma induction of rabbits with oral spray of Epstein-Barr virus-related herpesvirus from Si-IIA cells (HTLV-II-transformed Cynomolgus cell line): a possible animal model for Epstein-Barr virus infection and subsequent virus-related tumors in humans

Pathol Int. 1997 Jul;47(7):442-8. doi: 10.1111/j.1440-1827.1997.tb04522.x.

Abstract

Malignant lymphoma (ML) was induced in eight of nine rabbits inoculated by oral spray of the cell-free pellets from Si-IIA culture (HTLV-II-transformed leukocyte cell line of the Cynomolgus-producing Epstein-Barr virus (EBV)-related herpesvirus) after 64-141 days. None of the rabbits inoculated with EBV from B-95-8 cells or HTLV-II from MOT cells developed ML. Malignant lymphomas were usually of diffuse, large-cell or mixed type. HTLV-II infection was excluded by the polymerase chain reaction (PCR) and the particle agglutination test. EBV-encoded RNA-1 and EBV-related DNA were detected in the tumor tissues by in situ hybridization and PCR, respectively. Anti-viral capsid antigen of EBV antibody (anti-VCA) was observed 3 weeks after oral inoculation of Si-IIA cell-free pellets. Polymerase chain reaction revealed continuous detection of EBV-related virus DNA in the peripheral blood leukocytes from 3 days after oral inoculation. These results show that ML induced orally with Si-IIA cell-free pellets was caused by EBV-related herpesvirus harbored by Si-IIA cells. Oral spray of EBV from B-95-8 also induced EBV infection in rabbits, which was confirmed both by the presence of anti-VCA and by PCR. These oral infection and malignant lymphoma induction systems of rabbit using EBV-related virus from Si-IIA or human EBV are useful animal models for the study of EBV infection and EBV-related lymphomas in humans.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Administration, Oral
  • Animals
  • Antigens, Viral / immunology
  • Capsid Proteins*
  • Disease Models, Animal
  • Female
  • Herpesviridae Infections / virology*
  • Herpesvirus 4, Human / immunology
  • Herpesvirus 4, Human / pathogenicity*
  • Humans
  • In Situ Hybridization
  • Injections, Intraperitoneal
  • Injections, Subcutaneous
  • Liver / pathology
  • Liver / virology
  • Lymphoma / virology*
  • RNA, Viral / metabolism
  • Rabbits
  • Spleen / pathology
  • Spleen / virology
  • Tumor Virus Infections / virology*

Substances

  • Antigens, Viral
  • Capsid Proteins
  • Epstein-Barr viral capsid antigen
  • Epstein-Barr virus encoded RNA 1
  • RNA, Viral