Suppressive effects of tranilast on the expression of inducible cyclooxygenase (COX2) in interleukin-1beta-stimulated fibroblasts

Biochem Pharmacol. 1997 Jun 15;53(12):1941-4. doi: 10.1016/s0006-2952(97)00187-1.

Abstract

We investigated the effects of tranilast on inducible cyclooxygenase (COX2)-mediated prostaglandin E2 (PGE2) production and enzyme induction in interleukin-lbeta (IL-1beta)-stimulated cultured dermal fibroblasts. IL-1beta enhanced PGE2 production in cultured fibroblasts. Tranilast did not affect constitutive cyclooxygenase (COX1) or COX2 activity in non-stimulated or IL-lbeta-stimulated fibroblasts. However, the COX2 expression induced by IL-1beta was inhibited by tranilast. This result, that IL-1beta-induced COX2 expression was suppressed by tranilast, was confirmed by immunohistochemical analysis. Thus, it is possible for tranilast to regulate PGE2 production by inhibiting COX2 induction.

MeSH terms

  • Anti-Allergic Agents / pharmacology*
  • Cells, Cultured
  • Cyclooxygenase 2
  • Dinoprostone / biosynthesis*
  • Enzyme Induction / drug effects
  • Fibroblasts / drug effects
  • Fibroblasts / enzymology
  • Humans
  • Immunohistochemistry
  • Interleukin-1 / antagonists & inhibitors*
  • Isoenzymes / biosynthesis*
  • Keloid / prevention & control
  • Membrane Proteins
  • Prostaglandin-Endoperoxide Synthases / biosynthesis*
  • ortho-Aminobenzoates / pharmacology*

Substances

  • Anti-Allergic Agents
  • Interleukin-1
  • Isoenzymes
  • Membrane Proteins
  • ortho-Aminobenzoates
  • Cyclooxygenase 2
  • PTGS2 protein, human
  • Prostaglandin-Endoperoxide Synthases
  • tranilast
  • Dinoprostone