Molecular mechanisms of tazarotene action in psoriasis

J Am Acad Dermatol. 1997 Aug;37(2 Pt 3):S18-24.

Abstract

Psoriasis is a chronic immune-mediated disease that is characterized by the hyperproliferation and abnormal differentiation of keratinocytes and by inflammation. The epidermal changes associated with psoriasis may be due to the infiltration of inflammatory T lymphocytes and the release of cytokines in response to antigenic stimulation. Tazarotene is a retinoic acid receptor-specific retinoid with demonstrated efficacy in the topical treatment of psoriasis. Tazarotene down-regulates markers of keratinocyte differentiation, keratinocyte proliferation, and inflammation. The drug also up-regulates three novel genes TIG-1 (tazarotene-induced gene-1), TIG-2, and TIG-3, which may mediate an antiproliferative effect. The effect of tazarotene on these markers is probably a direct effect on gene expression rather than an indirect effect associated with disease improvement.

Publication types

  • Review

MeSH terms

  • Cell Differentiation / drug effects
  • Cell Division / drug effects
  • Dermatologic Agents / pharmacology*
  • Dermatologic Agents / therapeutic use
  • Humans
  • Keratinocytes / cytology
  • Keratinocytes / drug effects
  • Nicotinic Acids / pharmacology*
  • Nicotinic Acids / therapeutic use
  • Prodrugs / pharmacology*
  • Prodrugs / therapeutic use
  • Psoriasis / drug therapy*
  • Psoriasis / genetics
  • Psoriasis / physiopathology
  • Retinoids / pharmacology*
  • Retinoids / therapeutic use
  • Skin / drug effects

Substances

  • Dermatologic Agents
  • Nicotinic Acids
  • Prodrugs
  • Retinoids
  • tazarotene