Cell-mediated immunity after pertussis vaccination and after natural infection

Dev Biol Stand. 1997:89:307-14.

Abstract

The aim of this study was the investigation of the specific cell-mediated (CMI) responses induced by DTaP and to compare these data to immunity after natural infection. The ability of peripheral blood T-lymphocytes to respond to the pertussis related antigens pertussis-toxin (PT), filamentous haemagglutinin (FHA), and Pertactin (PRN) was investigated in 40 children before vaccination and at different times after vaccination with DTaP (Infanrix) by measurement of antigen-specific proliferation, lymphocyte phenotype, cytokine production and expression of activation markers (CD25, HLADR). Similar investigations were performed in children 4-6 weeks after recovery from natural pertussis. DTaP created a specific T-cell-response to PT, FHA and PRN that increased continually, depending on the progress of the vaccination schedule. In contrast to waning antibody titres, CMI was stable even between the post-basic vaccination and the pre-booster period. The magnitude of CMI after DTaP equalled those after natural infection. Measurement of cytokine-pattern showed induction of IFN-tau-producing T-helper-1-cells with lower stimulation of IL 10-producing T-helper-2-cells for DTaP and natural infection. Our data indicate that DTaP-vaccination induces a potent immune response to PT, FHA, and PRN at least equivalent to CMI after natural infection. The finding of a preferential T-helper-1-induction after DTaP and natural infection suggests a role of IFN gamma-activated macrophages in the protective response against B. pertussis-induced disease.

MeSH terms

  • Adhesins, Bacterial / immunology
  • Antibodies, Bacterial / biosynthesis
  • Antigens, Bacterial / immunology
  • Bacterial Outer Membrane Proteins / immunology
  • Cytokines / biosynthesis
  • Diphtheria-Tetanus-Pertussis Vaccine / immunology*
  • Diphtheria-Tetanus-acellular Pertussis Vaccines
  • HLA-DR Antigens / metabolism
  • Hemagglutinins / immunology
  • Humans
  • Immunity, Cellular
  • Infant
  • Lymphocyte Activation
  • Pertussis Toxin
  • Phenotype
  • Receptors, Interleukin-2 / metabolism
  • T-Lymphocytes / immunology*
  • Virulence Factors, Bordetella / immunology
  • Whooping Cough / immunology*
  • Whooping Cough / prevention & control

Substances

  • Adhesins, Bacterial
  • Antibodies, Bacterial
  • Antigens, Bacterial
  • Bacterial Outer Membrane Proteins
  • Cytokines
  • Diphtheria-Tetanus-Pertussis Vaccine
  • Diphtheria-Tetanus-acellular Pertussis Vaccines
  • HLA-DR Antigens
  • Hemagglutinins
  • Receptors, Interleukin-2
  • Virulence Factors, Bordetella
  • filamentous hemagglutinin adhesin, Bordetella pertussis
  • pertactin
  • Pertussis Toxin