Abstract
Drug resistance remains a significant obstacle to improving therapeutic outcome following treatment for malignant lymphoma. Eliminating P-glycoprotein as one mechanism of drug resistance may select for alternative, non-P-glycoprotein mechanisms of drug resistance. Understanding these alternative forms of drug resistance is imperative in order to improve therapy for NHL.
MeSH terms
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ATP Binding Cassette Transporter, Subfamily B, Member 1 / metabolism
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ATP-Binding Cassette Transporters / metabolism*
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Antineoplastic Agents / pharmacokinetics
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Apoptosis
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DNA Repair
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DNA Topoisomerases, Type II / metabolism
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Drug Resistance, Multiple
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Drug Resistance, Neoplasm*
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Humans
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Lymphoma, Non-Hodgkin / drug therapy
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Lymphoma, Non-Hodgkin / metabolism*
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Neoplasm Proteins / metabolism*
Substances
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ATP Binding Cassette Transporter, Subfamily B, Member 1
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ATP-Binding Cassette Transporters
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Antineoplastic Agents
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Neoplasm Proteins
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DNA Topoisomerases, Type II