Divergent transcriptional regulation among expanding human immunodeficiency virus type 1 subtypes

J Virol. 1997 Nov;71(11):8657-65. doi: 10.1128/JVI.71.11.8657-8665.1997.

Abstract

The current AIDS pandemic represents the uneven spread of multiple genetically related subtypes (A to J) of human immunodeficiency virus type 1 (HIV-1). Notably, HIV-1 E in southeast Asia and HIV-1 C in sub-Saharan Africa are expanding faster and are likely of greater global significance than the HIV-1 B subtype prevalent in the United States and Europe. While many studies have focused on genetic variation among structural genes, we chose to conduct a comparative analysis of the long terminal repeats of HIV-1 E and HIV-1 C isolates and report subtype-specific differences in enhancer copy numbers and sequences, as well as divergent activation in response to the cellular transcriptional activators Rel-p65 and NFATc and viral Tat. This study is the first to identify functional distinctions in promoter architecture between HIV-1 subtypes and raises the possibility that regulatory divergence among the subtypes of HIV-1 has occurred. Divergent transcriptional regulation may explain some of the epidemiologically observed differences in transmission and pathogenesis and underscores the need for further comparative analysis of HIV-1 regulation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • Binding Sites
  • DNA, Viral / genetics
  • DNA-Binding Proteins / metabolism
  • DNA-Binding Proteins / physiology
  • Gene Expression Regulation, Viral
  • Gene Products, tat / physiology
  • HIV Long Terminal Repeat
  • HIV-1 / classification
  • HIV-1 / genetics*
  • Humans
  • Molecular Sequence Data
  • NF-kappa B / physiology
  • NFATC Transcription Factors
  • Nuclear Proteins*
  • Phylogeny
  • Promoter Regions, Genetic
  • Proviruses
  • Sequence Alignment
  • Sequence Homology, Nucleic Acid
  • Transcription Factor RelA
  • Transcription Factors / metabolism
  • Transcription Factors / physiology
  • Transcription, Genetic
  • tat Gene Products, Human Immunodeficiency Virus

Substances

  • DNA, Viral
  • DNA-Binding Proteins
  • Gene Products, tat
  • NF-kappa B
  • NFATC Transcription Factors
  • Nuclear Proteins
  • Transcription Factor RelA
  • Transcription Factors
  • tat Gene Products, Human Immunodeficiency Virus

Associated data

  • GENBANK/AF023426
  • GENBANK/AF023427
  • GENBANK/AF023428
  • GENBANK/AF023429
  • GENBANK/AF023430
  • GENBANK/AF023431
  • GENBANK/AF023432
  • GENBANK/AF023433
  • GENBANK/AF023434
  • GENBANK/AF023435
  • GENBANK/AF023436
  • GENBANK/AF023437
  • GENBANK/AF023438
  • GENBANK/AF023439
  • GENBANK/AF023440
  • GENBANK/AF023441