T-cell receptor V beta-element expression in peripheral nerves of Lewis rats suffering from experimental autoimmune neuritis

J Neuroimmunol. 1997 Oct;79(1):69-75. doi: 10.1016/s0165-5728(97)00112-4.

Abstract

In experimental autoimmune neuritis (EAN), peripheral nerves are infiltrated by T-lymphocytes and macrophages. By RT-PCR and sequence analysis we characterized TCR V beta-element usage in sciatic nerve tissue of Lewis rats suffering from EAN induced by immunization with peripheral myelin antigens. Several TCR V beta-chain sequences were detected, which did not show homology to sequences of P2-reactive T cells published so far. In EAN induced with peripheral nerve myelin, but not with P2-protein or P2 peptide aa 53-78, TCR V beta 8.2 sequences identical to sequences of encephalitogenic myelin basic protein (MBP) reactive T-cells were identified. These results provide further evidence for a contribution of MBP-directed T-cell reactivity to the pathogenesis of myelin induced EAN and may have implications for the pathogenesis of human demyelinating neuropathies.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autoimmune Diseases / metabolism*
  • Autoimmune Diseases / pathology
  • Base Sequence
  • Female
  • Gene Amplification
  • Immunohistochemistry
  • Lymphocytes / metabolism
  • Molecular Sequence Data
  • Neuritis / metabolism*
  • Neuritis / pathology
  • Peptide Fragments / genetics
  • Peptide Fragments / metabolism*
  • Rats
  • Rats, Inbred Lew
  • Receptors, Antigen, T-Cell, alpha-beta / genetics
  • Receptors, Antigen, T-Cell, alpha-beta / metabolism*
  • Sciatic Nerve / metabolism*
  • Sciatic Nerve / pathology
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / physiology

Substances

  • Peptide Fragments
  • Receptors, Antigen, T-Cell, alpha-beta
  • T-cell receptor Vbeta 8.2