Abstract
A high fasting plasma homocysteine level is an independent risk factor for atherosclerosis and venous thrombosis. Vitamin therapy can lower homocysteine levels, but no benefit has yet been demonstrated; studies using clinical outcomes as endpoints are now in progress.
MeSH terms
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Arteriosclerosis / epidemiology
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Arteriosclerosis / etiology*
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Arteriosclerosis / therapy
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Folic Acid / therapeutic use
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Homocysteine / biosynthesis
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Homocysteine / blood*
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Humans
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Prognosis
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Pyridoxine / therapeutic use
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Risk Factors
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Thrombophlebitis / epidemiology
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Thrombophlebitis / etiology*
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Thrombophlebitis / therapy
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Vitamin B 12 / therapeutic use
Substances
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Homocysteine
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Folic Acid
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Pyridoxine
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Vitamin B 12