Cyclosporin A-sensitive release of Ca2+ from mitochondria in intact thymocytes

FEBS Lett. 1997 Nov 24;418(1-2):131-4. doi: 10.1016/s0014-5793(97)01361-6.

Abstract

Release of Ca2+ from mitochondria into cytosol in intact thymocytes was studied using the fluorescent dye Fluo-3. It was shown that the release of Ca2+ induced by the dithiol cross-linking agent phenylarsine oxide or by uncoupler was strongly inhibited by cyclosporin A, a specific inhibitor of the permeability transition pore (PTP) in mitochondria. Oxidative stress sensitized the pore so even partial uncoupling caused rapid cyclosporin A-sensitive release of Ca2+. The experiments on digitonin-permeabilized cells confirmed that uncoupling induced opening of the PTP, which forms the major pathway for rapid release of Ca2+ from thymocyte mitochondria.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium / metabolism*
  • Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone / pharmacology
  • Cells, Cultured
  • Cyclosporine / pharmacology*
  • Intracellular Membranes / drug effects
  • Intracellular Membranes / metabolism
  • Kinetics
  • Mitochondria / drug effects
  • Mitochondria / metabolism*
  • Oxidative Stress
  • Permeability
  • Rats
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / metabolism*
  • Thymus Gland

Substances

  • Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone
  • Cyclosporine
  • Calcium